Antibodies & immunoassays for small molecules 
Cancer cells aggressively reprogram amino acid metabolism to sustain their rapid proliferation and survival. By doing so, they deplete the […]
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Ten years ago, we introduced the first research ELISA kits designed to easily measure Kynurenine (Kyn) and Tryptophan (Trp) concentrations […]
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Our range of ELISA kits for quantifying tryptophan metabolites welcomes a new addition: Melatonin. This highly sensitive ELISA kit enables […]
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Anxiety and depression, autism, cognitive decline, gut motility and intestinal inflammation disorders, … Abnormal serotonin levels are detected in a […]
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On June 19th, Immusmol hosted a webinar about midbrain organoid models of Parkinson’s disease. Prof. Jens Christian Schwamborn (LCSB / OrganoTherapeutics) […]
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Dopamine, Serotonin, GABA, Glutamate, Histamine, Norepinephrine, Serine, Octopamine, … To generate spatial information about key neuromodulators and neurotransmitters, neurobiologists typically perform stainings against enzymes (e.g. TH for dopamine, GAD65-67 for GABA), receptors (e.g. Glutamate) or transporters (e.g. Norepinephrine, Glutamate). Why?
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TH expression does not necessarily indicate that neurons are metabolically active and produce dopamine. Therefore, several groups interested in organoid-based modeling of Parkinson’s disease have combined standard markers of dopaminergic neurons, such as TH and GIRK2, with direct staining of dopamine using anti-hapten antibodies, as well as ELISA kits for quantifying dopamine release in supernatant.
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